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Crucially, this signaling is bidirectional: central neuroendocrine outputs (e.g., via autonomic nerves and the HPA axis) reciprocally modulate gut physiologyaltering intestinal transit time, epithelial barrier permeability, and nutrient availabilitythereby reshaping the microbial metabolic environment and SCFA production capacity

No difference was observed with the weighted UniFrac analysis (Fig

Nuclear receptors as drug targets for metabolic disease

Spinal surgery to remove the degenerated disc and fuse the adjacent spinal segments may cause degeneration and instability of the neighboring discs (Karppinen et al., 2011)