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4 Conclusion Currently available therapeutics for the treatment of POAG are both limited and ineffective, rendering certain populations (e.g., non-responders to IOP-lowering treatments and those with advanced neurodegeneration) defenseless amidst a significant gap in care (Doucette et al., 2015)

Targeting MTH1 Transcription-Translation Process : Developing inhibitors, such as non-coding RNAs, to inhibit MTH1 expression and its tumor-promoting effects

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Amphetamines can also increase the release of excess glutamate from nerve endings of the CNS into the cortex, resulting in an imbalance of cortical signaling