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Similarly, we demonstrate here that VEGFAinduced CNV and degeneration of the RPE and photoreceptors can be potently inhibited by genetically inactivating NLRP3 inflammasome components or Il1r1 in VEGFA hyper mice

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It modulates the levels of ferroptosis-related signaling pathways, including ACSL4, SLC7A11, GPX4, and FTH1, and reduces the expression of MMP3 and MMP13, which could be one of the important mechanisms and pathways underlying its inhibitory effect on RA bone destruction