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The authors commented that these results suggest that T4 is a potent wound healing factor with multiple activities...

What exists is a real, documented biological mechanism that offers a plausible explanation, and a much larger body of anecdotal reporting built on top of that mechanism

Senescent cells, despite their pro-inflammatory SASP, also suppress tumor progression in some contexts through 'senescence surveillance'nearby immune cells recognize and destroy newly senescent cells, including pre-malignant cells undergoing oncogene-induced senescence

This characteristic makes it potentially safer for long-term applications, as elevated IGF-1 levels associated with traditional GH therapy raise concerns about increased cancer risk and metabolic complications