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Collectively, these observations highlight a convergent pathogenic mechanism in NDDs, in which mitochondrial quality control failure, through impaired mitophagy, defective protein import, and ROS-driven dysfunction, links mitochondrial damage, mtDNA-mediated inflammation, and protein aggregation [68,69,70,71,72,73]

Yuli Guo et al

Hence, an assessment of acetylcarnitine, PCr, and Pi time courses during a single exercise-recovery experiment increases the volunteers compliance with the experimental procedures and could improve potential correlation between the metabolites

Apoptotic cells typically upregulate the glycolytic pathway to provide additional energy, and mitochondria also participate in regulating apoptosis