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Concomitant with the spectrometric data, the immuno-expression of GPx4, a key inhibitor of ferroptosis, was significantly decreased (Figure 2B) while the staining level of FACL4, a protein that promotes ferroptosis by enhancing lipid peroxidation, was remarkably increased following CSD (Figure 2E)

Bortezomib overcomes cell-adhesion-mediated drug resistance through downregulation of VLA-4 expression in multiple myeloma