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BPC 157 also counteracts tumour cachexia and muscle wasting, characterised by increased pro-inflammatory/pro-cachectic cytokines such as interleukin-6 and tumour necrosis factor-, and significantly corrects deranged muscle proliferation and myogenesis through changes in the expression of FoxO3a, p-AKT, p-mTOR and P-GSK-3 (mitigating cancer cachexia)[3]

In adipose tissue (where NNMT is highly expressed), inhibition activates the polyamine flux pathway and shifts adipocytes toward a 'beige' phenotype with elevated energy expenditure

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These patients had previously (over 1 year ago) undergone transforaminal PELD