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demonstrated in enteric glial cell models that PS-NPs accelerate the aggregation of mutant A53T -synuclein, disrupt mitochondrial membrane integrity, and impair lysosomal function, collectively exacerbating dysregulation of the mitochondrial-lysosomal axis 41

Nevertheless, in some cell types or cell lines, the inhibition of GPX4 cannot induce ferroptosis, suggesting the presence of alternative mechanisms distinct from GSH-dependent pathways (70)

Exosomes help restore cellular communication, balance inflammation, and support tissue recovery

Donors were informed about the study and gave a written consent prior blood donation for the study