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Oxidative stress promotes calcium influx into cells and redistributes intracellular calcium from the endoplasmic reticulum (ER) to the cytoplasm, mitochondria, and nucleus, which increases mitochondrial permeability transition, facilitates the release of pro-apoptotic factors such as cytochrome c, and activates calcium-dependent nucleases, proteases, and lipases [127, 128]

Pachymic acid impairs breast cancer cell invasion by suppressing nuclear factor-kappaB-dependent matrix metalloproteinase-9 expression

For instance, calcipotriol-loaded NLCs, especially when co-loaded with methotrexate (CAL-MTX-NLCs), demonstrated a significant improvement in skin targeting, with enhanced delivery of methotrexate (2.44.4 times) and reduced calcipotriol release, mitigating the risk of irritation (Lin et al