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It is noteworthy that an excessive dietary intake of methionine-rich animal protein is an additional cause of hyperhomocysteinemia detected both in the fasting state and after an oral methionine load, especially when tissue folate and cobalamin stores are intact

ROS can also damage nucleic acids and cause DNAprotein crosslinking, strand breaking, and alteration in purine and pyridine bass structures, resulting in DNA mutations [26]

Of all the TAGEs, AGE-2 and AA-AGE appear to be particularly neurotoxic