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Hence, cysteine modifications are not limited to the well-known structural role of disulfide bonds in proteins synthesized in the endoplasmic reticulum (ER), but participate in fundamental intra- and inter-cellular signaling pathways

16,22-24 Tezacaftor/ivacaftor is approved for individuals aged 6 years and older with a homozygous F508del mutation or one tezacaftor/ivacaftor-responsive CFTR mutation

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In brief, inhibition of TrxR augments the susceptibility of tumor cells to oxidative stress, bolsters the selective eradication of cancerous cells, and instigates the cascade of apoptosis, which has received widespread attention in recent years making TrxR a potential target for cancer therapy (191, 192)