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Conclusions In conclusion, our findings demonstrate that VNS alleviates myocardial injury by reducing oxidative stress, mitochondrial damage, and ferroptosis in cardiomyocytes resulting from hepatic I/R injury

Prolonged PM2.5 exposure elevates risk of oxidative stress-driven nonalcoholic fatty liver disease by triggering increase of dyslipidemia

Dietary Reference Intakes for Thiamin, Riboflavin, Niacin, Vitamin B6, Folate, Vitamin B12, Pantothenic Acid, Biotin, and Choline (National Academies Press, 1998)

These findings align with the results from the intracellular replication assays in phagocytic cells ( Figure 7 ) and support the hypothesis that pre-exposure to EPA induces a transient repression of essential virulence genes, which reverses over time