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In summary, SQOR can either protect the cardiovascular system by enhancing mitochondrial function and antioxidant capacity, or exacerbate chronic cardiac pathology by depleting H 2 S signaling

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For that reason it has been suggested that plasma GSH/GSSG and plasma cysteine/CySS redox states be used together as a measure of oxidative stress in vivo in humans, since the plasma GSH/GSSG redox potential may less sensitive to acute oxidative disturbances (156)

Inflammopharmacology 15 (2), 8489