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[DOI] [PubMed] [Google Scholar] 41.Salmern-Brcenas E.G., Martnez-Zayas A., Vargas-Meja M., Villegas-Seplveda N., Briseo-Daz P., Aguilar-Rojas A., Baos-Hernndez C.J., Torres-Rojas F.I., Antao-Arias R., Hernndez-Rivas R

To a certain extent, these findings 22 support the gradual ROS response theory by Hekimi and co-workers as they suggest that (i) a subtle increase in mitochondrial ROS, as a consequence of mtDNA mutagenesis, is sufficient to modify the signalling in somatic stem cells and to disrupt their homoeostasis, and (ii) premature ageing may be accounted for by a cell type specific ROS-signalling mechanism and not by a general increase in ROS damage and subsequent RC dysfunction

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