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Interestingly, the expressions of hsa-mir-122-5p, IL6, FGL1, ACADM, LEP, INS, CASP1, NLRP3, ACAT1, TNF, ELAVL1, IGFBP7, ADAM9, GDF15, ACADVL, ECH1, octanoylcarnitine (octanoylcarnit.), isoleucine, leucine, L-valine, 7-keto-8-aminopelargonic acid (7-keto-8-amino.), creatinine, decanoylcarnitine (decanoylcarnit.), and hexanoylcarnitine (hexanoylcarnit.) were found to be upregulated in DCM, while those of hsa-mir-30c-5p, hsa-mir-30d-5p, hsa-mir-22-3p, GSTM2, GPX3, GSTM3, ADIPOQ, HADH, PRDX2, PRDX1, SERPINA1, A2M, PRDX6, APOA1, APCS, NPPA, bilirubin, butyric acid (butyrate), alanine, glutamine, and cytidine triphosphate (ara-CTP) were found to be downregulated in DCM

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Additionally, it is also crucial to evaluate whether free covalent inhibitors can be eliminated by metabolic enzymes in vivo during the development of covalent inhibitors