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The short answer: research doesnt yet support this theory enough to say definitively this is or is not the case

When reproducible physiology and biochemistry persist across years, modalities, and care settings, the question stops being is anything wrong? and becomes a governance one: Who owns the trigger for reevaluation when signals persist but dont fit a textbook pathway and where is that ownership documented so cases dont stall between handoffs

No cardiovascular risks: The hypertension and cardiovascular events associated with EPO don't occur with ARA-290 because the pathways responsible aren't activated