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Therapy with the opioid antagonist naltrexone promotes mucosal healing in active Crohns disease: a randomized placebo-controlled trial

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In response to stress, an intricate molecular mechanism facilitated by sensor cysteines within Keap1 allows Nrf2 to escape ubiquitination, accumulate within the cell, and translocate to the nucleus, where it can promote its antioxidant transcription program