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Given the mechanism by which non-dihydropyridine calcium channel blockers like diltiazem and verapamil slow conduction and decelerate signal transmission through the AV node, why are these medications suited for controlling the ventricular rate in atrial fibrillation and atrial flutter

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Because of its ability to increase intestinal permeability, functional screening of Zot protein was conducted, leading to the discovery of a human intestinal Zot analogue, zonulin, which, like Zot, has been shown to reduce TEER of intestinal tissue (7)

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