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Exposure to bisphenol A correlates with early-onset prostate cancer and promotes centrosome amplification and anchorage-independent growth in vitro

While several mechanisms have been reported, including the release of reactive oxygen species (ROS), inflammatory response, apoptosis, mitochondrial dysfunction, and enhanced blood-brain barrier (BBB) permeability (Zhang et al., 2024b), these findings fail to fully explain the complex processes underlying the onset and progression of HACD