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225 Administration of the PRMT4 inhibitor TP-064 in a mouse model of atherosclerotic cardiovascular disease can induce a decrease in monocyte tumor necrosis factor- (TNF-) secretion, downregulate the gene expression of the glycogen metabolism-related protein G6pc in the liver, and reduce plasma triglyceride levels, exerting regulatory effects from inflammatory and metabolic pathways

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However, oral administration of SCFAs triggered M2 to M1 polarization and increased the M1 population by activating glycolysis in TAMs (244)

145 In a randomized, controlled, crossover trial, at the same dose, zinc carnosine was shown to significantly reduce the increase in intestinal permeability seen with heavy exercise