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189 Specifically, inhibition of NMDARs or the downstream protein CaMKII suppresses the formation of glutamatergic synapses through the activity of NLGN1, whereas inhibition of NLGN2 activity suppresses the formation of GABAergic synapses

As a result, an infiltrate consisting of lymphocytes, plasma cells, macrophages, and eosinophils forms

This transcriptional suppression of inflammatory programs creates a cellular environment favoring repair over continued immune activation, while GHK-Cu's antioxidant propertiesincluding blocking formation of reactive oxygen species, detoxifying lipid peroxidation products such as acrolein, and preventing tissue-damaging free iron releaseprotect regenerating tissues from oxidative damage that can perpetuate inflammatory cycles