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doi: 10.1024/0300-9831.73.3.207

Wild type -syn overexpression or mutation causes mitochondrial fragmentation, mitochondrial membrane potential reduction, mitochondrial axonal transport inhibition, reactive oxygen species (ROS) production, and Ca 2+ homeostasis imbalance at mitochondria-associated membranes (MAMs), see 2+ transients, suggesting that impairment of -syn and Ca 2+ cross-talk in MAMs is involved in the pathogenesis of PD ( 2+ levels in mitochondria (Zhang et al., 2022)