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This review discusses the molecular mechanisms leading to eNOS uncoupling and the proposed therapeutic approaches to prevent or reverse eNOS uncoupling and restore endothelial function as a possible strategy for CVD treatment

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Similarly, Cys depletion can trigger ferroptosis, mainly due to impaired GSH biosynthesis and defective Fe/S cluster biogenesis, which result in an iron starvation response, increased iron uptake, and subsequent ferroptotic cell death [79,80]