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10.1016/j.ejca.2018.03.012 Summary Keywords mTORC1, mitochondria, mitochondrial functions, cancer, therapy Citation de la Cruz Lpez KG, Toledo Guzmn ME, Snchez EO and Garca Carranc A (2019) mTORC1 as a Regulator of Mitochondrial Functions and a Therapeutic Target in Cancer

suggests these actions might involve the activation of hormone-sensitive lipase (HSL), an enzyme critical for hydrolyzing stored triglycerides into free fatty acids and glycerol, possibly mitigating adipocyte size

Under hypoxia, cell metabolism shifts away from mitochondrial OXPHOS to glycolysis for energy production, which is primarily mediated by activation of HIF-1 signaling