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6d) have been used to identify the core phenotype of Mn deficiency, which includes glycosylation defects (resembling type II CDG), mitochondrial dysfunction, intellectual disability, growth retardation, and cerebral atrophy 209,214,215,217

The existence of -lactamase combinations, lesions in the OmpC-like and OmpF-like porins, and mutations in PBP3-ftsI might explain why Enterobacterales isolates are resistant to C/T (Karlowsky et al., 2022)

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