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In the reduced state, GSH can react with CYS within proteins to maintain their reduced forms or with reactive oxygen species to neutralise them

single-agent docetaxel or vintafolide

Characterized by iron toxicity accumulation, phospholipid peroxidation and dysfunctional mitochondria, ferroptosis is intricately linked to the hepatic microenvironment, influencing and being influenced by factors such as iron homeostasis, glucose metabolism, lipid metabolism, amino acid metabolism, and the gut-liver axis

Notably, Rh2 demonstrated good biosafety profile and could mitigate doxorubicin-induced cardiotoxicity by targeting inflammatory damages [187]