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doi: 10.2147/NDS.S243097 [DOI] [Google Scholar] 27

36 These frameworks suggest that the pathophysiology of AD is characterized by phosphorylated tau accumulation and propagation, 31 heightened inflammatory responses, dysregulation of oxidative stress (related to mitochondrial dysfunction), 37 alongside a gradual decline in cholinergic function

The DAC molecule has a milder version of the same problem, not an exemption from it: at 5001,000 mcg twice weekly a 5 mg vial is consumed in 2.55 weeks, but at the 300400 mcg low end of its documented range the same vial takes 68 weeks also far outside a 12 week reconstituted window

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