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For example, DSIP may regulate the activity of intracellular calmodulin or affect the interaction between calcium ions and other signaling molecules in cells, thereby affecting cell metabolism and growth adaptability

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This stands in marked contrast to what has happened in different situations in which clinical trials were designed according to predictive biomarkers such as, e.g., trastuzumab in HER2-positive breast cancer patients

Some chelators, such as D-penicillamine, also cause direct neurotoxicity, likely by crossing the blood-brain barrier and disrupting copper-dependent central nervous system enzymes, impairing energy metabolism, and increasing oxidative stress [245, 246]