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At the same time, this should be accompanied by systematic assays to quantify the protection conferred by intervention strategies aimed at gut microbiota (e.g., inhibiting TMA-producing bacteria), targeting the TMA-TMAO metabolic axis or direct scavenge of TMAO on chondrocyte ferroptosis and OA progression

Serum glutathione peroxidase, xanthine oxidase, and superoxide dismutase activities and malondialdehyde levels in patients with Parkinson's disease

*Correspondence: Barbara Falquetto, [email protected] Disclaimer All claims expressed in this article are solely those of the authors and do not necessarily represent those of their affiliated organizations, or those of the publisher, the editors and the reviewers