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In addition, genetic polymorphisms on B-vitamin-dependent enzymes in OCM, can result in disruption of OCM pathways which can lead to adverse phenotypes, even if dietary folate and related B-vitamin intakes are adequate (Reference Newton-Cheh, Johnson and Gateva13)

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5.8 L-carnosine, CoQ10 and ASD Among the lipophilic nutraceuticals, Coenzyme Q10 (Q10) supplementation reduced markers of oxidative stress (Mousavinejad et al., 2018)

$120 $) engineered to optimize cell-autonomous migration and wound healing dynamics via G-actin binding