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Overexpression of TFEB leads to degradation of bulk autophagic substrates such as damaged mitochondria (Nezich et al., 2015), long-lived protein aggregates (Martini-Stoica et al., 2016), and lipid droplets (Settembre et al., 2013), indicating the crucial role of TFEB in organelle-specific autophagy, such as mitophagy and lipophagy (Settembre et al., 2013

Glutathione injections bypass the digestive system, allowing for direct absorption into the bloodstream

Autophagy sequesters damaged lysosomes to control lysosomal biogenesis and kidney injury

On the other hand, an increase in the expression of this protein was observed in HT-22 co-treated with H