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Glutamine promotes tumor development in many distinct manners, such as (i) supporting epigenetic modifications in cellular FLICE-like inhibitory protein (cFLIP) and resistance of pancreatic tumor cells to apoptosis [93]

209 Furthermore, considerable evidence shows that obesity-associated adipocytes in pancreatic ductal adenocarcinoma can secrete IL-1 to attract tumor-associated neutrophils (TANs), which subsequently activate pancreatic stellate cells and contribute to tumor growth

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