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doi: 10.31202/ecjse.963670 215

Additionally, glucagon appears to reduce food intake through mechanisms distinct from GLP-1, providing a second independent appetite suppression pathway

Uwaezuoke SN

Summary Atherosclerotic lesions initiate with endothelial cell dysfunction causing modification of apoB containing lipoproteins (LDL, VLDL, remnants) and infiltration of immune cells, particularly monocytes, into the subendothelial space (Figure 1)