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4,11,15 Inflammatory responses mediated by proinflammatory cytokines and pathways, such as nuclear factor kappa B (NF-B) and Toll-like receptor 4 (TLR4), worsen the condition by amplifying oxidative stress and hindering neuronal repair

As the data used in this study were publicly available summary statistics, ethical approval and informed consent were not required

For example, ionotropic glutamate receptors represent ligand-gated calcium channels while GABA receptors trigger calcium influx via voltage-gated calcium channels [94]

Statements Data availability statement The original contributions presented in the study are included in the article/supplementary material