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Therefore, we used a model of spinal cord injury that has many characteristics found in human spastic syndrome [42] and can be used long-term to provide a realistic model of spasticity development in the tail muscle

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This mutation changes the amino acid sequence of the encoded GPP enzyme (P92L) within a well-conserved -bulge of myo-inositol monophosphatases

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Understanding this phase as potentially positive rather than concerning helps maintain protocol adherence

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