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Collectively, our study demonstrates that SLC1A4 mutations lead to dysregulation of multiple solute carrier protein genes causing metabolic stress, excitability defects, and synaptic abnormalities, providing a molecular framework for understanding SLC1A4 -related neurodevelopmental disorders and identifying potential therapeutic targets

Future studies will measure PFOS partitioning in other organs of interest, such as the liver and kidney, to further understand lifestyle factor influences on PFOS distribution

The quality of raw sequence data was assessed using FastQC (Babraham Bioinformatics)