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Together, these data support the hypothesis that RSL-induced ferroptosis is a novel death phenotype distinct from apoptosis, various forms of necrosis and autophagy

In summary, this review shows that the intake of flavonoids could increase the proliferation of certain gut bacteria that attenuate CVDs, and the relationships among flavonoids, GM, and CVD could offer a new perspective to understanding the mechanisms underlying the effects of flavonoids on CVD

GW4869, a neutral sphingomyelinase inhibitor, has been shown to reduce EV release and mitochondrial content [416, 473, 627]

Protopanaxadiol and metformin synergistically inhibit estrogen-mediated proliferation and anti-autophagy effects in endometrial cancer cells