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Record all handling procedures, including reconstitution volume, date, and storage conditions, in your laboratory notebook for future reference and reproducibility

Oral treatment with Cu(II)(atsm) increases mutant SOD1 in vivo but protects motor neurons and improves the phenotype of a transgenic mouse model of amyotrophic lateral sclerosis

Supporting this, therapeutic strategies aimed at correcting deficiencies or restoring copper-dependent signaling have shown promise

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