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Collectively, these data establish that SYK deficiency disrupts glycolytic flux in SCs through coordinated suppression of glucose transporter expression and glycolytic enzyme activity (Fig

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Both the electrical potential difference and the proton concentration gradient that are set up provide the energy used to generate ATP as the protons are drawn back, thermodynamically downhill, into the mitochondrial matrix through the ATP synthase complex (Figure 1)

This state of chronic oxidative stress also wreaks havoc on the immune system, particularly regarding mast cells