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Ardissino D, Merlini PA, Savonitto S et al

To better understand oxidative stress mechanisms, we have exposed cultured endothelial cells and macrophages to condensed E-cigarette vapour (E-cigarette condensate) and acrolein

In addition to the aforementioned prospective treatments outlined for TNBC, ongoing efforts are concentrated on developing novel therapeutic approaches

First, systematic studies are lacking to definitively establish whether dysregulated ferroptosis constitutes a key mechanism driving lesion progression or regression