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The intestine-liver-lung axis in septic syndrome

In these cases, PTMs accumulate in cells and contribute significantly to fat accumulation [54], hepatocyte apoptosis, inflammation, fibrosis [51], and cirrhosis [37], by altering signaling pathways affiliated with liver disease progression [43, 66, 67] and by targeting proteins for ubiquitin-mediated proteolysis [59]

GOT-1, glutamate oxaloacetate transaminase 1