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Potential Pathogenic Mechanisms of Hyperhomocysteinemia Despite the large amount of epidemiologic data in favor of a relationship between hyperhomocysteinemia and increased risk for cardiovascular disease, the true mechanisms remain hypothetical because of a scarcity of experimental data

The alanine at position 2 of the peptide was substituted with Aminoisobuturic acid (Aib) to protect the molecule from DDP-IV inactivation, and a lactam bridge was introduced between amino acids 16 and 20 to stabilize the secondary structure to ensure glucagon receptor potency

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In contrast, vancomycin, which does not penetrate mammalian cells, had no effect