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doi: 10.2337/diabetes.54.suppl_2.S97 22 YangSZhaoMJiaS

[Abstract]2025 Jul 17:43:102164

They are made of a repeating DNA sequence that protects our vital genetic code from damage

[30] The extensivity of the -catenin interacting proteins complicates our understanding: -catenin may be directly phosphorylated at Ser552 by Akt, which causes its disassociation from cell-cell contacts and accumulation in cytosol, thereafter 14-3-3 interacts with -catenin (pSer552) and enhances its nuclear translocation