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Epigenetic modifications: basic mechanisms and role in cardiovascular disease (2013 Grover Conference Series)

Their hemodynamic parameters are uniformly worse, encompassing higher filling pressures, lower cardiac output, and elevated pulmonary vascular resistance

Noteworthy, deletion of GluN1 also causes reduced expression of the transcriptional coactivator PGC1-alpha, which is highly expressed in PV interneurons and is a positive regulator of the expression of genes implicated in mitochondrial function and antioxidant defence [131]

This proposal is of particular interest as it enables the direct detoxification of AFB 1 through reactions that may not involve cytochrome P450