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In a rat model of DOX-induced cardiotoxicity, it has been unequivocally demonstrated that DOX induces the production of ROS and transforming growth factor-beta (TGF-), thereby promoting the transformation of cardiac fibroblasts into myofibroblasts (Tian et al., 2024)

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This leads to disruptions in the transcription of proteins vital to the mitochondrial ETC, thus inducing malfunction and hindering ATP synthesis, which in turn might escalate ROS generation [113]

(b) at least two unique peptides per protein must be identified, with the p value adjusted using BenjaminiHochberg correction for a false discovery rate (FDR) set to 0.01