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The hPrdx6 at acidic condition is also observed to gain thermal resistance indicating the importance of aiPLA2 activity in various extremophiles

These mechanisms may be regulated by the orphan nuclear receptor NR4A1, suggesting the involvement of a gene-mediated pathway in the anti-fibrotic effects of luteolin (Ye et al., 2025)

[40] Most data supports a polygenic, epistatic model, meaning that the disorder is caused by two or more genes and that those genes are interacting in a complex manner