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In this study, the authors show that inhibition of GLUL suppresses tumor growth in vivo

To examine whether the decreased FASN levels could result from NOTCH inhibition, we assessed the level of Notch signaling by identifying the levels of NOTCH1, NOTCH2, and their most direct downstream effector, HES1 (43), in GSH-depleted livers

To this end, we expressed exogenous NRF2-FL in Hs578 cells and analyzed the transcriptome after 8 h of Cyss deprivation in comparison to cells grown in the presence of Cyss and to cells expressing GFP as a control and treated in the same manner (Fig