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Introduction Pulmonary fibrosis (PF) is a chronic, progressive, and fatal interstitial lung disease (ILD) associated with loss of alveolar gas exchange function and excessive deposition of extracellular matrix (ECM) [1], leading to irreversible progressive lung scar formation

The study revealed that, in the presence of mutant HTT, a significant portion of genes with altered expression exhibited pronounced changes in DNA methylation, particularly in regions with low CpG content known to undergo methylation changes due to neuronal activity

Currently, several drugs with fast track designation are being tested in phase III clinical trials, and many other drugs have moved into phase II clinical trials

Physiologically, ROS and RNS are detoxified by enzymatic systems such as catalase, thioredoxin, and superoxide dismutase [38]