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2.3 Dysregulation and failure of endogenous antioxidant defense mechanisms Under physiological conditions, the myocardium maintains redox homeostasis through a robust endogenous antioxidant defense system, comprising enzymatic components like SOD, Catalase (CAT), and Glutathione Peroxidases (GPxs), as well as non-enzymatic antioxidants such as glutathione (GSH) and thioredoxin (Kumar et al., 2022)

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Using laser microdissected hippocampal neurons extracted from McGill-R-Thy1-APP transgenic rats we found that intraneuronal amyloid beta (iA)-burdened neurons had increased expression of genes related to oxidative stress and DNA damage responses including Ercc2, Fancc, Sod2, Gsr , and Idh1