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Among others, actions of IGF-I on cardiac stem cells include antioxidant effects, upregulation of telomerase activity, a delay in replicative senescence[288], and migration and homing of cardiac stem cells facilitating neovascularization in damaged hearts[287]

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Revisiting the concept of drug-resistant epilepsy: A TASK1 report of the ILAE/AES joint translational task force

Results: FD involves loss of function mutations in SLC40A1 that impair the iron export efficiency of ferroportin leading to iron retention in reticuloendothelial cells and hyperferritinemia with normal TSAT